Most heart attacks are caused by non -productive plates that penetrate the entire coronary artery. There is no such thing such as “1-Vessel”, “2-Vessel disease” or “left main disease”. The atherosclerotic plaque is continuous in all coronary arteries of the victims of heart attack.
In angioplasty, it is a tiny balloon imported In a narrow coronary artery that feeds the heart to force it to open wider to improve blood flow. Were not put For testing a randomized controlled test by 1992, not only failed to prevent heart attacks, but also failed to show any benefit of survival. However, the researchers only followed patients for six months and included people with relatively small diseases who may not have been sick enough to benefit from the process. Enter The mass test. The researchers recorded those with a strong blockage high on the left anterior descending coronary artery-the widowmaker or the widow (from the coronary artery disease is also the number one murderer of women)-and followed them for years. The findings? There was no difference in the next mortality or heart attack rates. There were only about 200 patients in this test, however. Perhaps the benefit was so thin that a larger number of patients needed to tease the result. Enter The Rita-2 study, which randomized more than a thousand patients. The researchers really found a clear difference in the risk of future death and heart attack, but it was in the wrong direction. The angioplasty team suffered twice as much as those who were randomized to give up the surgery, as shown below at 1:18 in my video Why the angioplasty stents don’t work better.
That was all before the stents came in fashion, though. Instead of only balloon Above the artery, how to permanently insert a stent, a metal tube grid, to open the artery, as you can see here at 1:33 in my video; Sure, this should help.
Enter Mass-II test, which, again, saw no benefit after one year-but was not benefiting warning After five years or even ten years. Then came the trial of courage, which randomized Thousands of patients, and this also fell flat on his face.
They mainly used Naked metal stents, however, are not the newest “drugs” that release drugs late. And what is happening with high -risk groups, such as those diagnosed with diabetes and other more serious diseases or those that have 100 percent blocks the arteries after having Heart attack? In meta-analysis after meta-analysis, look In five tests with 5,000 patients, there were There is no decrease in death, heart attack or even angina pain. In ten tests with more than 6,000 patients, there was no benefit for survival, heart attacks or pain relief. Now, we’re in more than twelve big tests and nothing: no avail From angioplasty and stent. “In addition, multiple analyzes have not been able to identify a single high -risk subset that benefits …” How is this possible? Naturally open the blood flow.
The reason he doesn’t work It is that the majority of heart attacks in real life is caused by narrow less than 70 percent-that is, probably non-influential restrictive lesions “-so the plates in our arteries that kill us tend to be not the ones that limit blood flow. Appears below and at 3:21 in my video hectare Two atherosclerotic plates. The one rotated with green and with the “damage that restricts the flow” pushes from the blood flow so much that it can see on a painting and doctors can go after a stent.
The problem was resolved and life was saved, right? No, because it was the invisible (described in yellow down) that did not even prevent the flow of blood that would kill us throughout, as you can see here at 3:27.
Indeed, most heart attacks are caused by non -productive plates that do not even cut blood flow by 50 %, as shown below at 3:40 in my video.
There is an incorrect perception, a “clogged ratio of constant coronary heart disease [that] It was particularly difficult displace“In which cholesterol plates slowly and inevitably violate blood flow, eventually cut completely and cause a heart attack. When these pimples are popping, they cause the blood to the arteries to thrive in the area … Before rupture, these plates often do not limit the flow. Coronary intervention (PCI), “that is, in pottery and stents.
The tightest blockages are made up to mainly calcified and dense fibrous scar tissue. They can still push and kill us, but there are so many of the smallest lesions, which are hidden in terms. The way we imagine The coronary arteries are by painting. The X -rays are taken after a black dye dye is injected into the arteries, so we can only see plates that insert blood flow. So we take These types of Tip-of-the-Isceberg’s Tip-The-Isceberg illustrations, the point of which “is to emphasize that most of the atherosclerotic plaque in the coronary arteries does not look good from angiography”, as you can see below at 4:49 in my own video. To really understand what’s going on in the arteries of people, must series in autopsy. William Clifford Roberts is perhaps the most primary cardiovascular pathologist in the world. What has he learned after studying the coronary arteries for 50 years? After examining nearly 2,000 bodies, he learned that atherosclerosis is a systemic disease.
“In patients with deadly coronary heart disease … The quantity of plaque is huge. There is only 1 plate here, another plate there, with normal lumen [clean arteries] Between plates. The plates are constant! No 5 mm section is deprived of the plate “throughout the coronary artery. So Dr. Roberts says:” Isolated coronary heart disease is a myth. There are no such things as ‘1-Vessel’ and ‘2 vessel disease’. The plate is found in all the usual coronary arteries if they are in 1 of them. ”
Four main coronary arteries food The heart -the right coronary artery, the left -wing coronary artery, the coronary artery region and the left anterior descending coronary artery, as shown at 6:00 in my own video.
If we add By their lengths, this is about 11 inches (28 cm) coronary arteries, which can be cut into slices of about 50 inches (5 mm) for examination. Appears below and at 6:17 in my video It’s what looks: the plate is not rap over one or two slivers but in all coronary arteries. If we look at more than a thousand of these slices from dozens of patients who died of heart attacks, “there was no part of the plate.” Thus, it is no wonder that stenting open in a single area has no effect on heart attacks or death.
